Francisella Tularensis Bacteria

Many studies have been performed to identify and characterize the virulence factors that f.
Francisella tularensis bacteria. Tularemia is a disease caused by francisella tularensis and the symptoms depend on where the bacteria invade. Francisella tularensis is a highly infectious bacterium that causes the rapid and often lethal disease tularemia. It is nonspore forming nonmotile and the causative agent of tularemia the pneumonic form of which is often lethal without treatment it is a fastidious facultative intracellular bacterium which requires cysteine for growth. Because of the highly infectious nature f.
Francisella tularensis is a pathogenic species of gram negative coccobacillus an aerobic bacterium. The bacterium was developed into an aerosol biological weapon by several countries in the past. Tularensis is currently classified as category a agent of bioterrorism. Tularemia is a zoonotic disease caused by the bacterium francisella tularensis.
Properties of francisella tularensis. Tularensis uses to infect a wide variety of hosts and host cell types evade immune defenses and induce severe disease and death. The species was originally discovered in squirrels in tulare county in 1911 the county for which the bacterium is named. This review focuses on the virulence factors that are.
The disease can be transmitted to humans through contact with infected animals such as the european brown hare lepus europaeus and ticks as vectors. Francisella tularensis is a gram negative bacteria and the agent causing tularemia also called rabbit fever. Due to its low infectious dose ease of spread by aerosol and high. The aim of this study was to isolate f.
Infections in the lungs cause high fever dry cough chest pains and swollen lymph nodes. Tularensis is considered to be a serious potential bioterrorist threat because it is one of the most infectious pathogenic bacteria known inhalation of as few as 10 organisms can cause disease and it has substantial capacity to cause serious illness and death.